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Monoamine oxidase and catechol-O-methyltransferase are largely answerable for the inactivation of catecholaminesThe outcome introduced below exhibit that, from the CNS of mice, the increased spillover of catecholamines due to not enough α2A- or α2C-adrenoceptors is accompanied by a better uptake from the catecholamine precursor L-DOPA and by an i